### Dr. SARTORI Cesium therapy cure for Cancer: page 1/3 http://www.mwt.net/~drbrewer/highpH.htm
An Introduction to Cesium Therapy, H. E. SARTORI, M.D.
Pharmacology, Biochemistry & Behavior. Vol. 21. Suppl. I, pp. 7 – 10. 1984. © Ankho International Inc.
Life Science Universal Medical Center, Suite 306 4501 Connecticut Avenue, Washington, DC 20008
A brief overview on the relevance of dietary factors in both development and prevention of cancer is presented. The pharmacologic properties of various food ingredients are discussed. Establishing of a special diet for the cancer patient is suggested. In addition, avoidance of certain foods is recommended to counteract mucus production of cancer cells. Evaluation of the nutrient content of certain diets in regions with low incidence of cancer has advanced the use of certain alkali metals, i.e., rubidium and cesium, as chemotherapeutic agents. The rationale for this approach, termed the “high pH” therapy, resides in changing the acidic pH range of the cancer cell by cesium (or rubidium) towards weak alkalinity in which the survival of the cancer cell is endangered, and the formation of acidic and toxic materials, normally formed in cancer cells, is neutralized and eliminated.
Antioxidants-Minerals-Cesium-Oxygenation-Diet-Ozone-Essential fatty acids-Potassium-Fish liver oils-Rubidium-Germanium-Sodium-Magnesium-Vitamins
ONE to 1.3 million cases of new cancer are anticipated in USA for the year 1984. Cancer mortality is second only to cardiovascular disease as the most common cause of death in the USA and in most
European countries. Squamous and basal cell skin cancer may contribute about 400,000 new cases, while lung and colorectal cancer may account for 139.000 cases each, compared to 116,000 for breast cancer of which 1.000 is projected for men. The remainder of cancers anticipated this year, in descending order of frequency, includes prostate, uterus, urinary, oral, pancreas, leukemias, ovary, and skin melanoma. The mortality of cancer is estimated for this year at approximately 185 cases per 100.000 of population or a total of 450,000. This is compared to 440.000, 431.000, and 423,000 deaths from cancers that have been reported for 1983, 1982 and 1981, respectively [ for 1984: 535,000; for 2004 (expected): 567,000].
Treatment modalities of cancer include surgery, radiation and chemotherapy. In those cases where surgery was successful the body’s own immune system was able to counteract the reduced amount of tumor tissue. Surgery is likely to produce metastasis without stimulation of the immune system. Radiation may cause damages to the organism, e.g., cancerogenicity of x-rays and other radiations, and their depression of the immune system is well known.
Prolonged chemotherapy may cause severe to lethal side effect in some instances, and the use of hormones and interferon are useful only for very few cases of cancer. Conventional treatments of cancer have produced 5 year survival rates which do not reflect the inexorable gradual deterioration of the cancer patients, nor their suffering from various treatment-related adverse reactions, and where chemotherapy of, e.g., breast cancer may shorten the life by 18 to 36 months v. no treatments at all. [See Appendix II: Summary of Adverse Effects]
Alternate cancer therapies include Orthomolecular/Eumetabolic approaches and Priore-radiation of cancer. The latter technique has been developed by Antoine Priore in France and has been described in detail elsewhere . In brief, it generates a combination of radiations in rotating plasmic solutions that are capable of penetrating living tissues for therapeutic purposes, without destroying biological systems.
The innovative techniques employed in the device since the early 1960’s have attracted scientific attention and research in the years following its introduction has demonstrated its efficacy in animal experiments, i.e., cure of cancer-bearing rats , attenuation of hypercholesterolemia in rabbits and survival of mice injected with fatal doses of Trypanosoma equiperdum, compared to corresponding controls. This approach is directed primarily towards a stimulation of the immune system rather than the cancerous cells themselves, resulting presumably in an acquired immunity which becomes intense and is even transferable by blood transfusion.
Dietary requirements which may lead to cancer have been generally ignored. Various diets and eating habits appear to bear some significance to susceptibility as well as to prevention and/or treatment of cancer. The macrobiotic-related dietary approach of Michio Kushi [4,5] for cancer treatment focuses on the use of whole grains, e.g., 50% to 60% of daily intake, with locally grown vegetables, e.g., 20% to 30%, excluding potatoes, tomatoes, peppers, eggplant, and especially tropical fruits. This is complemented by 10% to 15% of daily food consumption of beans and seaweeds in addition to 5% miso soup (& other soybean products) and about 5% supplemental foods. The main difficulty with this approach is the necessity of complete compliance. The possible association of specific diet ingredients to cancer development, treatment and/or prevention is outlined below which then leads to the rationale of the use of an alkali metal, i.e., cesium, as chemotherapeutic agent.
#NUTRITION AND CANCER MUCUS
The cancer cell is known to produce large amounts of mucus and this, in turn, shields the cancer cells from the immune system and from being penetrated by chemotherapeutic agents. It even protects against radiation if the layer of mucus is thickened up. Later this mucus can also be demonstrated in the blood, e.g., with the HLB blood test. Use of certain agents help to dissolve the blocking effect of the mucus.
This includes the use of beta-carotene which decomposes blocking mucoid proteins mediated by electrical charges. It gets inactivated and decolorized by the blocking mucoid. The necessary dosage for optimal effect produces the so-called “carotenemia”, an orange-yellow tinge to the patient’s skin. Likewise, heparin inactivates the immune repelling and immune binding capacities of the mucoid proteins by electrical charges. Furthermore, compounds like bromelain, papain (in green papayas), Wobemugos®, or pancreatic enzymes will not only break down mucus but also destroy leukemic cells.
TABLE I : CANCER PROTECTIVE NUTRIENTS WHICH MAY REDUCE INCIDENCE OF CANCER
Possible Mechanism of Action
– Cesium and Rubidium 1 :
Raise pH in cancer cells and gene repair.
– Omega-3 Essential Fatty Acids (Eicosapentaenoic acid and Docosahexaenoic acid):
Repair cell membranes; enhance the immune system and prostaglandin synthesis.
– Carotene and Vitamin A:
Decompose blocking mucus and enhance immune system.
Antioxidant and broadens electron donor capacity of cancer cell membrane.
– Vitamin C and Bioflavonoids:
Enhance the immune system.
Oxygen carrier; interferon stimulator, and gene repair.
– Vitamin D2 (Ergocalciferol):
Immune enhancer; precursor of tumosteron.
Membrane stabilizer; part of xanthine oxidase which mobilizes iron from liver and of aldehyde oxidase necessary for fat oxidation.
Electron donor; antioxidant, and immune stimulant.
Enzyme activator; gene and membrane stabilizer.
– Nitriles(amygdalin; mandelonitrile-o-glucuronide or Laetrile®)2:
Broaden electron donor capacity of the cancer cell membrane; release of cancer growth inhibiting benzaldehyde and cyanide.
– Allicin and sinigrin:
Natural cancer inhibitors.
Desalting substance that lowers sodium in cancer cells.
Precursor of dehydroepiandrosterone (DHEA) an anticancer, antiaging, and antiobesity factor.
Membrane and gene stabilizing.
– Photonic Energy:
Decomposes blocking mucus surrounding the cancer cells (are found in KlRLIAN positive raw vegetables and fresh vegetable juices, especially carrot juice.)
Slows down mitosis and the multiplication of cancer cells.
– Food Fiber:
Reduces passage-time, decreasing the exposure of intestines, especially the colon, to carcinogens; binds toxic substances and carcinogens.
– B-complex vitamins, especially Riboflavin (Vitamin B2):
Required for cell respiration (esp. B2) and catalysts for numerous enzymes.
– Vitamin E (mixed D-Tocopherols):
Antioxidant; membrane and gene stabilizer.
– Pantothenic acid:
Stimulates adrenals and DHEA-formation.
– Folic Acid:
Coenzyme with B 12 and C for protein utilization; carbon carrier for heme and nucleic acid formation: thus it normalizes gene formation of cancers. It also stimulates the production of hydrochloric acid.
– Certain Amino Acids (Cystein, Arginine, Ornithine; also IGF 1, etc., from Cornuparvum of the Chinese deer Cervus nippon/elaphus)
Stimulate the anticancer human growth hormone
*See text for the presence of these compounds in various food ingredients.
– 1 Rubidium is especially beneficial for pancreatic and liver cancers and other malignancies that present with clinical depression.
Rubidium chloride (RbCl) is established as an antidepressant of low toxicity and acts similar to the equally nontoxic lithium orotate.
– 2Amygdalin, found in bitter almonds, apricot and peach pits, is hydrolyzed to lmandelonitrile-o glucoside and oxidized with platinum black to mandelonitrile-ßglucuronide or Laetrile® which, in turn, is turned by a ß-glucuronidase into glucuronic acid, and the cancer-supressive HCN and benzaldehyde.
It is recommended, therefore, to avoid all mucus producing foods, e.g., all dairy products, except goat milk derivatives, egg whites (whereas soft yolks are highly beneficial) for all blood types (with possible exception of Type Bs), and, more specifically, all grains, except brown rice, for Type Os (and Bs), all meat, especially for Type As (and Abs) which do well on whole grains and locally grown vegetables, all soy bean (Glycine max) derivatives for Type Os and Bs (e.g., soy milk, tofu, tempe, etc.), tomatoes, except for Type Os, most beans and nuts, except almonds and walnuts, for Type Bs and Os (pumpkin seeds: good). Foods with high contents of sugar, alcohol, citrus fruits, refined carbohydrates, salt, pork in every form, as well as the blood-type specific mucus-producing foods mentioned previously, may also increase the specific mucus production of cancer cells.
The common (kosher) practice of not eating other items with milk is of significant bearing in this respect because (cow’s) milk produces a mucus that coats all the food, and thus prevents all the nutrients from being absorbed. We have also been using (CaNa2-)EDTA for chelation therapy which has been shown to reduce incidences of cancer and heart diseases by 90% and 50%, respectively . Likewise, increased cancer incidence may result from consumption of egg-whites and certain crustaceans, e.g., lobster, shrimp, and crayfish, due to their high content of nucleic acids which can be detrimental to the cancer patient. Therefore, the elimination of these items from diet may conceivably reduce the incidence of cancer.
# VITAMINS, ESSENTIAL FATTY ACIDS, AND FREE RADICALS
The Smithsonian Institute has done a study on the incidence of cancer in sharks. They examined 25,000 sharks and found only one individual case with cancer. This suggests that the shark (e.g., genus Squalus) is most probably immune to cancer. The shark liver oil contains vitamins and other compounds with anticancer activity. This includes squalene which is also contained in cod liver and in olive oil. Squalene increases the polarization of the cell membrane and thereby may facilitate the action of immune system on the cancer cell. This compound is also a precursor for dehydroepiandrosterone (DHEA) which possesses anticancer activity. Moreover, squalene has been implicated in the mechanism of Na accumulation by the cancer cell. For example, the high uptake of Na+ by the cancer cell induces an electrical potential that defies the immune mechanisms.
Thereafter, both, blood forming organs and the blood cells, show a high content of Na+ which can undergo desodification by squalene and certain sulphur-containing compounds also found in shark oil, such as taurine (oxidized sulfur-containing amine forming conjugates with cholic and deoxycholic acid in bile; also a CNS neurotransmitter or neuromodulator) and isethionic acid (HO.CH3.CH2.SO3H). Lithium orotate counteracts Na+ retention as well and also effectively increases the monocyte and granulocyte counts.
Certain vitamins, e.g., carotenes and Vitamin A, are associated with low incidence of lung cancer. The excess of other vitamins, i.e., over 5 g of vitamin C/day, may enhance tumor growth in leukemias, and over 10 g/day in lymphomas and similar cancers.
It appears that the foods with the most decisive effects on the reduction of cancer incidence in humans and in animals contain omega 3 unsaturated essential fatty acids (EFAs) which are also found in oils derived from linseed, chestnuts, beechnuts, soy beans, walnuts, and wheat germ. In addition, soybean and wheat germ oils contain high amounts of Vitamin E. Also rich in EFAs are cold climate legumes of which soybeans and their products, adzuki, black and pinto beans, and lentils are beneficial for blood type As, pinto and navy beans, lentils, and soybean products are beneficial for Type ABs, adzuki beans and black-eyed peas are beneficial for Type Os, and kidney and navy beans for Type Bs.
Richest in o-3 EFAs are cold water fish of which cod, halibut, swordfish, sturgeon, and herring are beneficial for Type Os, cod, mackerels, salmon, and trout for Type As, cod, salmon, halibut, mackerel, sardines, sturgeon and caviar for Type Bs, and cod, halibut, herrings, mackerels, tuna, and sturgeon for Type ABs. Cold-water plankton and sea vegetables are other rich sources of o-3 EFAs. In cold climate animals, eicosasapentaenoic acid (EPA) is immediate precursor of 3-series of prostaglandins which are involved in protection of cellular functions and prevent the formation of 2-series prostaglandins which are carcinogenic. EPA inhibits formation of thromboxane A2 (TxA2) an extremely potent platelet aggregator and vasoconstrictor and antagonist of prostacyclin that may contribute to cancer by local tissue hypoxia. A diet rich in EPA also decreases elevated levels of LDL cholesterol and triglycerides, and inhibits in vitro chemotactic and aggregating activities of neutrophils.
Careful selection of oils should be considered which should be kept refrigerated and kept under a nitrogen seal until sold to prevent oxidation due to high contents of o-3 EFAs in cod liver oil and other fish liver oils, and both o-3 and o-6 in linseed or flaxseed oil and wheat germ oil. Furthermore, in fish oils, the potential toxicity of Vitamins A and D should be considered. The use of margarine or hydrogenated oils, non-cold pressed polyunsaturated oils, shortening, bacon, grease, non-dairy creamers, egg substitutes, commercial mayonnaise and salad dressings, as well as of tropical oils like coconut oil, palm oil or cottonseed oil should be avoided. Excess amounts of polyunsaturated compounds may be oxidized which damages the cell membrane and these oxidized EFAs cannot be transported anymore into the mitochondria by carnitine and thus circulate in the bloodstream.
These oxidized EFAs promote the release of free radicals which are carcinogenic, may drive LDL cholesterol from the bloodstream into the liver and body cells, and destroy HDL cholesterol and interfere with the HDL-associated lecithin-cholesterol acyl transferase (LCAT). Note that terminal cancers consistently have HDL levels from 0.0 to 20.0 mg/dL and total cholesterol levels from 20.0 (!) to 120 mg/dL which seem to be the most reliable indicators of the life expectancy of the patient, and HDL levels under 30 mg/dL and total cholesterol under.150 mg/dL should prompt a workup to rule out a cancer. [For further details see Author’s Note under (3) at the end of this article].
Oxidized EFAs also cause a significant elevation of uric acid (indicating destruction of cellular nucleoprotein), cause iron deficiency and anemia, liver disease, intestinal damage and obstruction, amyloidosis (abnormal waxy deposits in tissues), hypertension, gallstones, and increase the incidence of atherogenesis (formation of artherosclerosis). Free radical induction from overheating fats is also due to formation of the highly toxic from decomposition of glycerin.
Note that acrolein is one of the degradation products of cyclophosphamide, used in cancer chemotherapy, and is thought to be the cause of hemorrhagic cystitis and bladder cancer in patients treated with cyclophosphamide. [See also Appendix III: Dietary Carcinogens]
In subtropical or tropical climates the use of “southern oils” is recommended. These include light and dark sesame oil, sunflower and safflower (not for Type Os, Bs, and ABs), and especially, virgin olive oil (beneficial to all blood types, as is walnut oil) which contain small amounts of the anticancer shark factor squalene. The foregoing observations suggest the use of cod liver oil (except for Type O nonsecretors) for several months to increase EPA intake, and to administer in addition 6000 U of Vitamin A and 600 U of Vitamin E. Other supplements will be wheat germ oil (not for Type Os) to provide energy from octacosanol and to improve physical fitness. Sesame oil may be advantageous against various bleedings, i.e., nose, gastrointestinal and gynecological bleedings.
The supplement EPA can be given as concentrate or by consumption of EPA – rich foods which may also be beneficial in certain heart, blood vessel, bowel, and immune diseases, as well as cancer. In temperate climates, the caloric intake from EFAs should be approximately 2% to 3%. Furthermore, gamma linolenic acid from evening primrose oil has been found, via synthesis of the series-1 prostaglandins, most notably prostaglandin E1 (PGE1), to decrease elevated LDL cholesterol levels, lower blood pressure by vasodilatation, inhibit thrombosis/platelet aggregation, and to normalize cancer cells.
# OXYGENATION (see Appendix I)
It is generally assumed that healthy cells resist becoming cancerous if they are provided with adequate nutrients. Insufficient supply of a given critical nutrient may lead to or facilitate cancer induction. The same is true of the amount of oxygen reaching the cells since low cellular oxygen levels may result in anaerobic conditions which will further cancer development. The lack of oxygen has long been suspected in carcinogenesis because it leads to an anaerobic metabolism where, essentially, glucose is converted into lactic acid and the pH in the cancer cells becomes acidic. The acidic pH because of lack of O2 may cause breakdown of RNA and DNA and damage the cellular control mechanisms involved.
The development of acidic toxins usually will lead to the destruction of cell structures. Therefore, reversing this condition requires adequate oxygenation. There are certain elements, e.g., germanium, which may prove beneficial for cellular oxygenation. Germanium possesses 8 valences and therefore can carry 4 atoms of oxygen and may provide the oxygenation needed for the cancer cell to evoke anticancer effect. Ginseng normally grows only on germanium rich soil and should provide a good source for this phenomenon. However, the use of soil antibiotics in homegrown ginseng may interfere in the production of an effective ginseng due to its lack of soil derived germanium. This sensitivity of cancer cells to oxygenation explains, in part, the effectiveness of oxidative therapies, such as hydrogen peroxide and, particularly, ozone, in certain cancers.
Ozone, especially directly I.V., in the author’s experience, without any doubt, greatly enhances the effects of all cancer therapies including the one suggested herein.
Certain antioxidant in food preservatives, e.g., butylated hydroxytoluene, butylated hydroxyanisole, and 5,6-benzoflavone may possess anticancer activity. Ornithine, arginine, and IGF1 and other amino acids stimulate/modulate human growth hormone secretion/action, and thus enhance the immune system, reverse uncontrolled cancer growth, and suppress prolactin production.
# INORGANIC COMPOUNDS AND ELEMENTS
The presence of certain elements in diet is as important as the choice of diet for reducing the incidence of cancer and even in the management of the cancer patient. For example, selenium deficiency has been associated with increased cancer incidence.
Other elements with some anticancer activity include molybdenum, zinc, magnesium, and germanium.
Brewer’s  analyses of reports dealing with certain regions of the world with low incidence of cancer has motivated his rationale for the high pH therapy for cancer. This was primarily based on the high amounts of cesium and rubidium found in the food consumed in these areas, most notably the Hopi Indian territory in Arizona, Hunza Land in Northern Pakistan, volcanic areas in Hawaii, Austria, and in selected regions of Kenya and Brazil. This represents a novel approach in cancer chemotherapy based on changes in cancer cell pH and possible inactivation of ionic hydrogen of the tumor cell to reduce acidity. This approach has been referred to as “high pH” therapy. A combined effect of low pH and high body temperature has been also suggested in cancer treatment and termed the “low pH” therapy. Both pH therapies are briefly outlined below; the low pH therapy was devised by Von Ardenne  and the high pH therapy by Brewer .
Both have been shown to be effective therapeutic measures for the treatment of cancer in laboratory animals and humans.
# LOW- pH Therapy
In this therapy, glucose is injected into the blood stream. As a consequence the cancer cell pH drops to the 5.5 range. The patient is then placed in a chamber heated from 45 to 50°C (113 to 122°F) for 4 to 6 hrs . Diathermy is also applied over the tumor area which, in the absence of a blood supply, will cause the temperature of the tumor mass to rise to over 55° (133°F). At these high temperatures, the life of cancer cells is observed to be very short. An apparent drawback to the therapy is that a case of severe toxemia may result because of leakage of acidic and toxic material from the tumor masses .[See “Hyperthermia” in Appendix]
# HIGH – pH Therapy
The rapid uptake of cesium and rubidium observed for cancer cells is the theoretical approach of high pH therapy . This therapy has been tested using CsCl or Cs2C03 in conjunction with the administration of ascorbic and retinoic acids, zinc and selenium salts.
The weak acids which are absorbed by the tumor cells have been shown to enhance the negative potential gradient across the membrane.
Zinc and selenium salts, when absorbed on the membrane surface, act as broad and moderately strong electron donors. These ions and salts have been shown in mice to drastically enhance the uptake of cesium and rubidium ions. For treatment of cancer patients the administration of 6 to 9 g of CsCl or CsCO3 for several days is believed to be tolerable and sufficient to raise the pH in the tumor cells to a weak alkaline level of approximately pH 8 where the life cancer cell is shortened. In addition, the presence of cesium and rubidium salts in the body fluid is expected to neutralize the acidic and toxic material emanating from the disintegrating tumor mass.
Results from both animal experiments, mainly through Messiha , and these of ours in limited clinical trial in humans in our clinic , are indicative of a high success rate of Cs treatment in cancer therapy.
Thus, both dietary factors and selected elements and vitamins may play a more significant role in the pathogenesis and pathology of certain cancers than has been previously accounted for. Moreover, changes in dietary habits may have a lasting effect on protection against cancer development and progression.
To conclude, the author presents a list of cancer protective nutrients and their main functions and importance outlined in Table I.
Appendix III lists “Naturally Occurring Dietary Carcinogens”.
The helpful assistance of Dr. Fathi S. Messiha in the preparation of this manuscript is gratefully acknowledged.
– 1. Blumer, W. and T. H. Reich. Leaded gasoline: a cause of cancer. Environ 1nt 3: 465-471. 1980.
– 2. Brewer, A. K. Mechanism of carcinogenesis: Comments on therapy. J Int Acad Prev Med 5: 29-53. 1979.
– 3. Graille, J.-M. Dossier Priore, une Nouvelle Affaire Pasteur. Paris: Editions Denöel. 1984.
– 4. Kushi, Michio. Cancer and Heart Disease: The Macrobiotic Approach to Degenerative Disease. Tokyo: Japan Publications Inc. 1982.
– 5. Kushi, Michio. The Cancer Prevention Diet, Michio Kushi’s Nutrirional Blueprint for the Relief and Prevention of Disease. New York: St. Martin’s Press. 1983.
– 6. Messiha, F. S., A. EI-Domeiri, and H. F. Sproat. Effects of lithium and cesium salts on sarcoma-I implants in the mouse. Neurobehav Toxicol 1: 27-31. 1979.
– 7. Sartori, H. E. Cancer 1984. Orwellian or Eutopian. Washington.DC: Life Science Universal Medical Center. 1984.
– 8. Von Ardenne, M. Hyperthermia and cancer therapy. Oncol Chemother Pharmacol 4: 137-139. 1980.
# AUTHOR’S NOTE: Certain parts of the original manuscript that was submitted for publication were deleted by the editor of this journal as being too controversial or not sufficiently proven to warrant inclusion in this article. This slightly edited version restores the text of the original manuscript and in some places clarifies where all tooterse language may have obscured the intended meaning. Specific areas that at the time of the publication were too controversial include:
(1) Any reference to the therapeutic use of oxidative therapies, in particular, direct I.V. ozone.
(2) Any reference to any blood-type specific individual nutritional requirements as this, in 1984, was virtually unexplored, except for James D’Adamo in Portsmouth, N.H. and the author, and where the latter found that almost all the failures of vegetable only approaches to cancer, including Michio Kushi’s, were Type Os, whereas, generally, Type As responded to these therapies.
(3) Extremely low serum cholesterol levels (in mg/dL) as the most reliable indicators for the survival potential of terminal cancer patients, e.g., 0.0 HDL &
Blood Type A (and AB) High Cancer Risk Profile:
(1) Thomsen-Friedenreich [Tn (GalNAc…) & T (Galo1o3GalNAc…)] antigens/epitopes, found in ~90% of all cancers/metastases, B-cell lymphomas (human lymphotropic virus Type II-induced), & multiple myeloma, resemble the Type A antigen (GalNAc…). This impairs the ability of the immune system of Type As (& ABs) to recognize malignant cells and also weakens NK cell activity. Thus, Type As are at increased risk for most cancers, except bladder cancers where Os and Bs at the highest risk, pancreas, liver, & esophagus cancers where only Os are low risk, whereas Os in melanomas and Bs in bone cancers are at the highest risk . Note that the specific lectin of the escargot snail, Helix pomatia, unmasks T & Tn antigens in (pre)cancerous cells. Regular intake of escargots is recommended nutritional treatment for all Type As (& ABs) as cancer-preventative and may help reverse breast cancers and lymphomas.
(2) Type As with metastatic cancer show elevated fibrinogen, factor VIII, and von Willebrand factor (vWF) which attach to an aberrant platelet glycoprotein that, in turn, attaches to cancer cells and thus causes metastasis. Heparin and other blood thinners effectively prevent this type of metastasis.
(3) Type As have the greatest tendency to manifest a “cancer personality” which is explained in “Cesium Therapy in Cancer Patients”, infra, in these proceedings.
Pharmacology, Biochemistry & Behavior. Vol. 21. Suppl. I. pp. 11-13. 1984. © Ankho International Inc.